Core Clinical Cases in Medicine and Medical Specialties Second Edition: A problem-solving approach
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You've read your textbook and your course notes. Now you need to apply your knowledge to real-life clinical situations.
The problem-solving approach of Core Clinical Cases guides you to think of the patient as a whole, rather than as a sequence of unconnected symptoms. With its emphasis on everyday practice strongly linked to underlying theory, the series integrates your knowledge with the realities of managing clinical problems, and provides a basis for developing sound analytical and confident decision-making skills.
The core areas of undergraduate study are covered in a logical sequence of learning activities; the same questions are asked of each clinical case, followed by detailed explanatory answers. OSCE counselling cases, with related questions and answers, also feature in each section.
Key concepts and important information are highlighted, and the reader-friendly layout reflects exactly the type of question you will encounter, making these volumes the perfect revision aid for all types of case-based examination.
The Medicine and Medical Specialties volume, in which all medical subjects have been brought together in a single volume for this second edition, focuses on the following topics: diabetes, endocrinology, rheumatology, renal medicine, cardiology, care of elderly people, respiratory medicine, gastroenterology, haematology, oncology emergencies; neurology, infectious diseases, and dermatology.
Volumes in the Core Clinical Cases series remain absolutely invaluable in the run up to clinical, written, or OSCE examinations, and ideal course companions for all undergraduate medical students at various stages in their clinical training.
noted by the clinicians at that time that the central nervous system (CNS) disorder had presented with pharyngitis. This led to the possibility of a post-infectious autoimmune CNS disorder similar to Sydenham’s chorea, in which group A betahaemolytic streptococcal antibodies cross-react with the basal ganglia and result in abnormal behaviour and involuntary movements. Anti-streptolysin-O titres have subsequently been found to be elevated in the majority of these patients. It seemed possible that
‘long-term remissions’ in the future and a reduction in the prevalence of patients suffering the consequences of persistent hypercortisolaemia, principally osteoporosis and the risks associated with impaired glucose tolerance or frank diabetes mellitus, hypertension and obesity. Cushing’s disease 61 OSCE counselling cases OSCE COUNSELLING CASE 2.5 – ‘Will “the tumour” spread to other parts of my body?’ Metastatic spread of pituitary tumours has been documented but is extremely rare. The word
conditions that the patient has that predispose to arrhythmias, e.g. thyrotoxicosis or valvular heart disease. A drug history is very important because some drugs may precipitate arrhythmias or influence the management of the arrhythmia (e.g. verapamil must not be given to patients who are on beta-blockers because it may result in circulatory collapse). A2: What is the likely diagnosis? The likely diagnosis is atrial fibrillation (AF) secondary to thyrotoxicosis as a result of the history of
bladder resulting from detrusor muscle instability (urge incontinence), pelvic floor incompetence (stress incontinence), neurogenic bladder (e.g. spinal cord lesions, cerebrovascular disease), incontinence associated with dementia, prostatism or bladder tumour or stone, and immobility from any cause. Assessment must include a full history and examination, including per rectum and (if appropriate) per vagina examinations, and CNS and locomotor assessments. Investigations include urinalysis, MSU,
weight loss is marked. The technical options to assess the glycaemic response are near-patient (usually self-) testing of blood glucose and laboratory testing (HbA1c levels). Home blood glucose monitoring is commonly taught to patients in this scenario, but critics argue that it is of little value and is very expensive. Unless a patient or the clinician is going to manipulate therapy based only on the results of HBGM (and this is unlikely to be the case here), one can argue that it should not be